Browsing Biochemistry (Scholarly Publications) by Author "DAVEY, GAVIN"
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Age-related changes in H2O2 production and bioenergetics in rat brain synaptosomes
DAVEY, GAVIN (2008) -
Alternatively folded proteins with unexpected beneficial functions.
DAVEY, GAVIN (2012) -
Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.
DAVEY, GAVIN; BOLAND, JOHN; ROWAN, MICHAEL (2015) -
Complex I and energy thresholds in the brain
DAVEY, GAVIN (2008) -
Complex I is rate limiting for oxygen consumption in the nerve terminal.
DAVEY, GAVIN (2009) -
Decylubiquinone increases mitochondrial function in synaptosomes
DAVEY, GAVIN (2010) -
Depletion of glutathione does not affect electron transport chain complex activities in brain mitochondria: Implications for Parkinsons's disease and postmortem studies
DAVEY, GAVIN (2011)Glutathione is an important antioxidant in the brain that appears to be decreased, in conjunction with mitochondrial complex I activity, in Parkinson?s disease patients. In postmortem analysis, measurement of glutathione ... -
Fc gamma receptors: Glycobiology and therapeutic prospects
DAVEY, GAVIN; HAYES, JERRARD (2016) -
Galactosyltransferase 4 is a major control point for glycan branching in N-linked glycosylation
MCDONALD, ANDREW; HAYES, JERRARD; DAVEY, GAVIN; TIPTON, KEITH (2014)Protein N-glycosylation is a common post-translational modification that produces a complex array of branched glycan structures. The levels of branching, or antennarity, give rise to differential biological activities for ... -
GlycoForm and Glycologue: two software applications for the rapid construction and display of N-glycans from mammalian sources.
DAVEY, GAVIN; MCDONALD, ANDREW; TIPTON, KEITH (2010)Background: The display of N-glycan carbohydrate structures is an essential part of glycoinformatics. Several tools exist for building such structures graphically, by selecting from a palette of symbols or sugar names, ... -
High-level inhibition of mitochondrial complexes III and IV is required to increase glutamate release from the nerve terminal
GLUCHOWSKA, SONIA AGNIESZKA; DAVEY, GAVIN (2011)Background: The activities of mitochondrial complex III (ubiquinol-cytochrome c reductase, EC 1.10.2.2) and complex IV (cytochrome c oxidase EC 1.9.3.1) are reduced by 30-70% in Huntington's disease and Alzheimer's ... -
Partial inhibition of complex I activity increases Ca2+-independent glutamate release rates from synaptosomes
DAVEY, GAVIN; Kilbride, Seán M.; Telford, Jayne E.; Tipton, Keith F. (2008)Mitochondria have been implicated in the pathogenesis of several neurodegenerative disorders and, in particular, complex I (NADH:ubiquinone oxidoreductase, EC 1.6.5.3) activity has been shown to be partially reduced in ... -
Synergizing metabolic flux analysis and nucleotide sugar metabolism to understand the control of glycosylation of recombinant protein in CHO cells.
DAVEY, GAVIN (2011)BACKGROUND: The glycosylation of recombinant proteins can be altered by a range of parameters including cellular metabolism, metabolic flux and the efficiency of the glycosylation process. We present an experimental set-up ...