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Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.

DAVEY, GAVIN; BOLAND, JOHN; ROWAN, MICHAEL (2015)

A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

DAVEY, GAVIN (2016)

miR-223: Infection, inflammation and cancer

O'NEILL, LUKE (2013)

Toll-like receptor 4 (TLR4) down-regulates microRNA-107 increasing macrophage adhesion via cyclin-dependent kinase 6

O'NEILL, LUKE; SHEEDY, FREDERICK (2011)

Queuosine deficiency in eukaryotes compromises tyrosine production through increased tetrahydrobiopterin oxidation.

KELLY, VINCENT; BERNSTEIN, IIANA; RAKOVICH, TATSIANA (2011)

Mapping the interactions between a RUN domain from DENND5/Rab6IP1 and sorting nexin 1.

KHAN, AMIR; FRANKLIN, EDWARD (2012)

Blocking retinoic acid receptor-α enhances the efficacy of a dendritic cell vaccine against tumours by suppressing the induction of regulatory T cells

GALVIN, KAREN; HIGGINS, SARAH; MILLS, KINGSTON; STEFANSKA, ANNA; MORAN, BARRY; DUNGAN, LARA (2013)

Altered Expression of Caspases-4 and -5 during Inflammatory Bowel Disease and Colorectal Cancer - Novel Diagnostic and Therapeutic Potential.

MILLS, KINGSTON; CREAGH, EMMA (2015)

Immunosuppressive networks and checkpoints controlling antitumor immunity and their blockade in the development of cancer immunotherapeutics and vaccines.

MILLS, KINGSTON (2013)

The MyD88+ Phenotype Is an Adverse Prognostic Factor in Epithelial Ovarian Cancer.

O'NEILL, LUKE; GLEESON, NOREEN; O'TOOLE, SHARON; O'BYRNE, KEN; O'LEARY, JOHN; SMYTH, PAUL (2014)

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Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.

A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

miR-223: Infection, inflammation and cancer

Toll-like receptor 4 (TLR4) down-regulates microRNA-107 increasing macrophage adhesion via cyclin-dependent kinase 6

Queuosine deficiency in eukaryotes compromises tyrosine production through increased tetrahydrobiopterin oxidation.

Mapping the interactions between a RUN domain from DENND5/Rab6IP1 and sorting nexin 1.

Blocking retinoic acid receptor-α enhances the efficacy of a dendritic cell vaccine against tumours by suppressing the induction of regulatory T cells

Altered Expression of Caspases-4 and -5 during Inflammatory Bowel Disease and Colorectal Cancer - Novel Diagnostic and Therapeutic Potential.

Immunosuppressive networks and checkpoints controlling antitumor immunity and their blockade in the development of cancer immunotherapeutics and vaccines.

The MyD88+ Phenotype Is an Adverse Prognostic Factor in Epithelial Ovarian Cancer.

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This Academic/Research Unit

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Subject

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Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.

A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

miR-223: Infection, inflammation and cancer

Toll-like receptor 4 (TLR4) down-regulates microRNA-107 increasing macrophage adhesion via cyclin-dependent kinase 6

Queuosine deficiency in eukaryotes compromises tyrosine production through increased tetrahydrobiopterin oxidation.

Mapping the interactions between a RUN domain from DENND5/Rab6IP1 and sorting nexin 1.

Blocking retinoic acid receptor-&#945; enhances the efficacy of a dendritic cell vaccine against tumours by suppressing the induction of regulatory T cells

Altered Expression of Caspases-4 and -5 during Inflammatory Bowel Disease and Colorectal Cancer - Novel Diagnostic and Therapeutic Potential.

Immunosuppressive networks and checkpoints controlling antitumor immunity and their blockade in the development of cancer immunotherapeutics and vaccines.

The MyD88+ Phenotype Is an Adverse Prognostic Factor in Epithelial Ovarian Cancer.

Blocking retinoic acid receptor-α enhances the efficacy of a dendritic cell vaccine against tumours by suppressing the induction of regulatory T cells