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Nutrient ingestion increased mTOR signaling, but not hVps34 activity in human skeletal muscle after sprint exercise.

MURRAY, JAMES (2013)

Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.

DAVEY, GAVIN; BOLAND, JOHN; ROWAN, MICHAEL (2015)

A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

DAVEY, GAVIN (2016)

Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1 beta and IL-6

CUNNINGHAM, COLM; SKELLY, DONAL; MURRAY, CAROL (2011)

A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1beta, TNF-alpha and IL-6 Challenges in C57BL/6 Mice.

CUNNINGHAM, COLM (2013)

At the extreme end of the psychoneuroimmunological spectrum: delirium as a maladaptive sickness behaviour response

CUNNINGHAM, COLM (2013)

Prior pathology in the basal forebrain cholinergic system predisposes to inflammation-induced working memory deficits: reconciling inflammatory and cholinergic hypotheses of delirium.

CUNNINGHAM, COLM (2012)

Delirium is a strong risk factor for dementia in the oldest-old: a population-based cohort study.

CUNNINGHAM, COLM (2012)

New horizons in the pathogenesis, assessment and management of delirium.

CUNNINGHAM, COLM (2013)

Systemic inflammation and delirium: important co-factors in the progression of dementia

CUNNINGHAM, COLM (2011)

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Nutrient ingestion increased mTOR signaling, but not hVps34 activity in human skeletal muscle after sprint exercise.

Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.

A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1 beta and IL-6

A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1beta, TNF-alpha and IL-6 Challenges in C57BL/6 Mice.

At the extreme end of the psychoneuroimmunological spectrum: delirium as a maladaptive sickness behaviour response

Prior pathology in the basal forebrain cholinergic system predisposes to inflammation-induced working memory deficits: reconciling inflammatory and cholinergic hypotheses of delirium.

Delirium is a strong risk factor for dementia in the oldest-old: a population-based cohort study.

New horizons in the pathogenesis, assessment and management of delirium.

Systemic inflammation and delirium: important co-factors in the progression of dementia

Browse

All of TARA

This Academic/Research Unit

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Author

Subject

Date Issued

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Nutrient ingestion increased mTOR signaling, but not hVps34 activity in human skeletal muscle after sprint exercise.

Amyloid Oligomers and Mature Fibrils Prepared from an Innocuous Protein Cause Diverging Cellular Death Mechanisms.

A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1 beta and IL-6

A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1beta, TNF-alpha and IL-6 Challenges in C57BL/6 Mice.

At the extreme end of the psychoneuroimmunological spectrum: delirium as a maladaptive sickness behaviour response

Prior pathology in the basal forebrain cholinergic system predisposes to inflammation-induced working memory deficits: reconciling inflammatory and cholinergic hypotheses of delirium.

Delirium is a strong risk factor for dementia in the oldest-old: a population-based cohort study.

New horizons in the pathogenesis, assessment and management of delirium.

Systemic inflammation and delirium: important co-factors in the progression of dementia