Browsing by Author "HAWI, ZIARIH"
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Absence of the 7-repeat variant of the DRD4 VNTR is associated with drifting sustained attention in children with ADHD but not in controls
JOHNSON, KATHERINE ANNE; GILL, MICHAEL; ROBERTSON, IAN H; CONNOR, THOMAS JOSEPH; HAWI, ZIARIH (2008) -
Association between dopamine transporter (DAT1)genotype, left sided inattention, and an enhanced response to methylphenidate in attention deficit hyperactivity disorder
GILL, MICHAEL; HAWI, ZIARIH; ROBERTSON, IAN; FITZGERALD, MICHAEL (2005)A polymorphism of the dopamine transporter gene (DAT1, 10-repeat) is associated with attention-deficit hyperactivity disorder (ADHD) and has been linked to an enhanced response to methylphenidate (MPH). One aspect of the ... -
DNA variation in the SNAP25 gene confers risk to ADHD and is associated with reduced expression in prefrontal cortex.
HAWI, ZIARIH; GILL, MICHAEL (2013) -
Dopa decarboxylase gene polymorphisms and attention deficit hyperactivity disorder (ADHD): no evidence for association in the Irish population
GILL, MICHAEL; MCCARRON, MARY; HAWI, ZIARIH; FITZGERALD, MICHAEL (2001)Dopa decarboxylase (DDC) is an enzyme which catalyses the decarboxylation of both dopa to dopamine and L-5 hydroxytryptophan to serotonin. Both catecholamines are major neurotransmitters of the mammalian nervous system. ... -
Dopaminergic system genes in ADHD: toward a biological hypothesis
GILL, MICHAEL; HAWI, ZIARIH; MCCARRON, MARY (2002)Converging evidence has implicated abnormalities of dopamine neurotransmission to the pathology of attention deficit hyperactivity disorder (ADHD). Several genetic association studies have been published, but so far, no ... -
Epistasis between neurochemical gene polymorphisms and risk for ADHD
GILL, MICHAEL; HAWI, ZIARIH (2011)A number of genes with function related to synaptic neurochemistry have been genetically associated with attention deficit/hyperactivity disorder. However, susceptibility to the development of common psychiatric disorders ... -
Evidence that variation at the serotonin transporter gene influences susceptibility to attention deficit hyperactivity disorder (ADHD): analysis and pooled analysis.
GILL, MICHAEL; HAWI, ZIARIH (2002)Reduced central serotonergic activity has been implicated in poor impulse regulation and aggressive behaviour in animals, adults and also young children.1,2 Two recently published studies have implicated variation at a ... -
fMRI activation during response inhibition and error processing: the role of the DAT1 gene in typically developing adolescents and those diagnosed with ADHD
JOHNSON, KATHERINE; BARRY, EDWINA; ROBERTSON, IAN; GARAVAN, HUGH; HAWI, ZIARIH; MULLIGAN, AISLING; GILL, MICHAEL (2011)The DAT1 gene codes for the dopamine transporter, which clears dopamine from the synaptic cleft, and a variant of this gene has previously been associated with compromised response inhibition in both healthy and clinical ... -
Genome-wide analysis of copy number variants in attention deficit hyperactivity disorder: the role of rare variants and duplications at 15q13.3.
ANNEY, RICHARD; GILL, MICHAEL; HAWI, ZIARIH (2012) -
Joint Analysis of the DRD5 Marker Concludes Association with Attention-Deficit/Hyperactivity Disorder Confined to the Predominantly Inattentive and Combined Subtypes
GILL, MICHAEL; HAWI, ZIARIH (2004)Attention-deficit/hyperactivity disorder (ADHD) is a highly heritable, heterogeneous disorder of early onset, consisting of a triad of symptoms: inattention, hyperactivity, and impulsivity. The disorder has a significant ... -
Preferential Transmission of Paternal Alleles at Risk Genes in Attention Deficit/Hyperactivity Disorder
GILL, MICHAEL; SEGURADO, RICARDO; GALLAGHER, LOUISE; HAWI, ZIARIH; FITZGERALD, MICHAEL (2005)Family, twin, and adoption studies have demonstrated a significant genetic contribution to the etiology of attention-deficit/hyperactivity disorder (ADHD). Pharmacological, neuroimaging, and animal-model findings suggest ... -
Serotonergic System and Attention Deficit Hyperactivity Disorder (ADHD): a potential susceptibility locus at the 5-HT1B receptor gene in 270 Trios from a multi-center sample
GILL, MICHAEL; HAWI, ZIARIH; FITZGERALD, MICHAEL (2002)Attention deficit hyperactivity disorder (ADHD) is a highly heritable and heterogeneous disorder, which usually becomes apparent during the first few years of childhood. Imbalance in dopamine neurotransmission has been ... -
Spatial attentional bias as a marker of genetic risk, symptom severity and stimulant response in ADHD.
JOHNSON, KATHERINE; ROBERTSON, IAN; HAWI, ZIARIH; GILL, MICHAEL; FITZGERALD, MICHAEL (2007)Attention-deficit hyperactivity disorder (ADHD) is a heritable childhood onset disorder that is marked by variability at multiple levels including clinical presentation, cognitive profile, and response to stimulant ... -
Synaptosomal-Associated Protein 25 (SNAP-25) and Attention Deficit Hyperactivity Disorder (ADHD): Evidence of linkage and Association in the Irish Population.
GILL, MICHAEL; HAWI, ZIARIH; FITZGERALD, MICHAEL (2002)Several lines of evidence have suggested that ADHD is a polygenic disorder produced by the interaction of several genes each of a minor effect. Synaptosomal-associated protein 25 (SNAP-25) is a presynaptic plasma membrane ...