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A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings.

ANNEY, RICHARD; GILL, MICHAEL (2011)

Home environment: association with hyperactivity/impulsivity in children with ADHD and their non-ADHD siblings

GILL, MICHAEL; ANNEY, RICHARD (2011)

Genome-wide analysis of copy number variants in attention deficit hyperactivity disorder: the role of rare variants and duplications at 15q13.3.

ANNEY, RICHARD; GILL, MICHAEL; HAWI, ZIARIH (2012)

Home environment: association with hyperactivity/impulsivity in children with ADHD and their non-ADHD siblings.

ANNEY, RICHARD (2013)

The one and the many: effects of the cell adhesion molecule pathway on neuropsychological function in psychosis

HARGREAVES, APRIL; GILL, MICHAEL; MORRIS, DEREK; ANNEY, RICHARD; CORVIN, AIDEN; DONOHOE, GARY (2013)

Individual common variants exert weak effects on the risk for autism spectrum disorderspi.

ANNEY, RICHARD; MERIKANGAS, ALISON; HERON, ELIZABETH; GILL, MICHAEL; GALLAGHER, LOUISE (2012)

A novel approach of homozygous haplotype sharing identifies candidate genes in autism spectrum disorder.

ANNEY, RICHARD; MERIKANGAS, ALISON; MCGRATH, JANE; GALLAGHER, LOUISE (2012)

Linkage to chromosome 1p36 for attention-deficit/hyperactivity disorder traits in school and home settings.

ANNEY, RICHARD; GILL, MICHAEL (2008)

A genomewide scan for common alleles affecting risk for autism.

ANNEY, RICHARD; GALLAGHER, LOUISE; GILL, MICHAEL; BOLSHAKOVA, NADIA; SEGURADO, RICARDO; HERON, ELIZABETH; MCGRATH, JANE; TANSEY, KATHERINE; BRENNAN, SEAN; MERIKANGAS, ALISON; MERIKANGAS, ALISON KATHLEEN (2010)

Gene-ontology enrichment analysis in two independent family-based samples highlights biologically plausible processes for autism spectrum disorders.

GILL, MICHAEL; GALLAGHER, LOUISE; ANNEY, RICHARD; KENNY, ELAINE (2011)

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A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings.

Home environment: association with hyperactivity/impulsivity in children with ADHD and their non-ADHD siblings

Genome-wide analysis of copy number variants in attention deficit hyperactivity disorder: the role of rare variants and duplications at 15q13.3.

Home environment: association with hyperactivity/impulsivity in children with ADHD and their non-ADHD siblings.

The one and the many: effects of the cell adhesion molecule pathway on neuropsychological function in psychosis

Individual common variants exert weak effects on the risk for autism spectrum disorderspi.

A novel approach of homozygous haplotype sharing identifies candidate genes in autism spectrum disorder.

Linkage to chromosome 1p36 for attention-deficit/hyperactivity disorder traits in school and home settings.

A genomewide scan for common alleles affecting risk for autism.

Gene-ontology enrichment analysis in two independent family-based samples highlights biologically plausible processes for autism spectrum disorders.

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All of TARA

This Academic/Research Unit

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A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings.

Home environment: association with hyperactivity/impulsivity in children with ADHD and their non-ADHD siblings

Genome-wide analysis of copy number variants in attention deficit hyperactivity disorder: the role of rare variants and duplications at 15q13.3.

Home environment: association with hyperactivity/impulsivity in children with ADHD and their non-ADHD siblings.

The one and the many: effects of the cell adhesion molecule pathway on neuropsychological function in psychosis

Individual common variants exert weak effects on the risk for autism spectrum disorderspi.

A novel approach of homozygous haplotype sharing identifies candidate genes in autism spectrum disorder.

Linkage to chromosome 1p36 for attention-deficit/hyperactivity disorder traits in school and home settings.

A genomewide scan for common alleles affecting risk for autism.

Gene-ontology enrichment analysis in two independent family-based samples highlights biologically plausible processes for autism spectrum disorders.