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A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

DAVEY, GAVIN (2016)

Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1 beta and IL-6

CUNNINGHAM, COLM; SKELLY, DONAL; MURRAY, CAROL (2011)

Systemic inflammation induces acute working memory deficits in the primed brain; relevance for delirium

CUNNINGHAM, COLM (2012)

Queuosine deficiency in eukaryotes compromises tyrosine production through increased tetrahydrobiopterin oxidation.

KELLY, VINCENT; BERNSTEIN, IIANA; RAKOVICH, TATSIANA (2011)

High-level inhibition of mitochondrial complexes III and IV is required to increase glutamate release from the nerve terminal

GLUCHOWSKA, SONIA AGNIESZKA; DAVEY, GAVIN (2011)

Evaluating the role of Toll-like Receptors in diseases of the Central Nervous system

Bowie, Andrew; Carty, Michael (2011)

A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1beta, TNF-alpha and IL-6 Challenges in C57BL/6 Mice.

CUNNINGHAM, COLM (2013)

At the extreme end of the psychoneuroimmunological spectrum: delirium as a maladaptive sickness behaviour response

CUNNINGHAM, COLM (2013)

Dose-related effects of vitamin D on immune responses in patients with clinically isolated syndrome and healthy control participants: study protocol for an exploratory randomized double- blind placebo-controlled trial.

WALSH, CATHAL; FLETCHER, JEAN (2013)

Prior pathology in the basal forebrain cholinergic system predisposes to inflammation-induced working memory deficits: reconciling inflammatory and cholinergic hypotheses of delirium.

CUNNINGHAM, COLM (2012)

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A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1 beta and IL-6

Systemic inflammation induces acute working memory deficits in the primed brain; relevance for delirium

Queuosine deficiency in eukaryotes compromises tyrosine production through increased tetrahydrobiopterin oxidation.

High-level inhibition of mitochondrial complexes III and IV is required to increase glutamate release from the nerve terminal

Evaluating the role of Toll-like Receptors in diseases of the Central Nervous system

A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1beta, TNF-alpha and IL-6 Challenges in C57BL/6 Mice.

At the extreme end of the psychoneuroimmunological spectrum: delirium as a maladaptive sickness behaviour response

Dose-related effects of vitamin D on immune responses in patients with clinically isolated syndrome and healthy control participants: study protocol for an exploratory randomized double- blind placebo-controlled trial.

Prior pathology in the basal forebrain cholinergic system predisposes to inflammation-induced working memory deficits: reconciling inflammatory and cholinergic hypotheses of delirium.

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All of TARA

This Collection

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Subject

Date Issued

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A Knowledge-Based System for Display and Prediction of O-Glycosylation Network Behaviour in Response to Enzyme Knockouts.

Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1 beta and IL-6

Systemic inflammation induces acute working memory deficits in the primed brain; relevance for delirium

Queuosine deficiency in eukaryotes compromises tyrosine production through increased tetrahydrobiopterin oxidation.

High-level inhibition of mitochondrial complexes III and IV is required to increase glutamate release from the nerve terminal

Evaluating the role of Toll-like Receptors in diseases of the Central Nervous system

A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1beta, TNF-alpha and IL-6 Challenges in C57BL/6 Mice.

At the extreme end of the psychoneuroimmunological spectrum: delirium as a maladaptive sickness behaviour response

Dose-related effects of vitamin D on immune responses in patients with clinically isolated syndrome and healthy control participants: study protocol for an exploratory randomized double- blind placebo-controlled trial.

Prior pathology in the basal forebrain cholinergic system predisposes to inflammation-induced working memory deficits: reconciling inflammatory and cholinergic hypotheses of delirium.